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The paper presents an in-depth description of the use of vasopressors in the emergency setting from the nurses’ point of view. The subject matter was discussed in meetings of the “Emergency Nursing” Nursing Group, a Massachusetts General Hospital community-wide network composed of 80% “cluster nurses” from the Emergency Department and 20% nurses from the emergency medicine service. The cluster nurses serve as consultants and administrators and are responsible for organizing and directing nursing practice in the ED. The emphasis of the group is on nursing practice in complex care environments where the nurses strive to ensure optimal care for the patient. Through three specific clinical scenarios, the author provides an in-depth description of the use of vasopressors in the ED and the role that the “cluster nurse” plays in the implementation of this practice.Inhibition of cdc2 gene expression confers resistance to a benzo(a)pyrene-DNA adduct in Chinese hamster V-79 cells.
DNA damage response is considered an important step in the development of tumorigenesis following the exposure of cell to genotoxic agents. One of the molecular events related to activation of tumor suppressor genes and the inhibition of cell proliferation is inhibition of cdc2 gene expression. This process is controlled by a transcriptional factor, dTAF-2. We previously reported that exposure of Chinese hamster V-79 cells to the benzo(a)pyrene (BP)-DNA adduct alpha-hydroxy-BP-N7-guanine (BP-O6-Gua) led to decreased dTAF-2 protein levels, increased growth inhibition, and a decreased sensitivity of cells to TNF-alpha-induced cell death. We report here that the decreased cell sensitivity could be rescued by removal of BP-O6-Gua from the cells in association with a recovery in dTAF-2 protein levels and inhibition of cdc2 gene expression. To
